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This is why peptide therapy should begin with the patient’s goal and medical history

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Persistent pressure overload induces a cascade of pathological events: on one hand, sustained high pressure leads to insufficient myocardial perfusion—particularly in the subendocardial regions prone to ischemia—and strongly activates the renin–angiotensin–aldosterone system and the sympathetic nervous system, resulting in sodium and water retention, peripheral vasoconstriction, and myocardial fibrosis (65, 67)